62.5

Teruo Hidaka

Juntendo University

Teruo Hidaka's research has significantly advanced our understanding of podocyte biology and its role in kidney disease. Studies have identified key proteins, such as neurofilament heavy polypeptide and cathepsin D, that protect against podocyte injury and proteinuria, while others, like Rac1 and Sall1, promote glomerular repair and limit sclerosis. Hidaka's work has also shed light on the importance of sorting nexin 9 in endocytic processes within injured podocytes and the detrimental effects of cathepsin D in pathogenesis of proteinuria and chronic kidney disease. These findings hold great promise for developing novel therapeutic strategies to treat kidney diseases, particularly those affecting podocyte function and integrity.

Podocyte DepletionPodocyte BiologyPodocyte Development
Commercial signal 62.2
Scientific signal 64.8
Social signal 64.0
Papers 12
0 Patent-to-paper cites
211 Paper cites

scientifiq.ai is an experimental platform. The platform relies on open source data and it may contain errors. Its primary goal is to advance scientific research on innovation.